RCMI Coordinating Center (RCMI CC) Header Logo

Search Result Details

This page shows the details of why an item matched the keywords from your search.
One or more keywords matched the following properties of Das, Salil
PropertyValue
overview I have broad background in biochemistry, molecular biology and environmental toxicology, with specific training and expertise in several research areas. My current research focuses on molecular mechanisms of lung injury/fibrosis, lung cancer and breast cancer. I have developed a guinea pig model of lung injury/fibrosis induced by direct and environmental smoke and mustard gas, a chemical warfare agent and currently investigating the potential role of an antioxidant liposome containing N-acetylcysteine and tocopherol as an antidote against lung injury/fibrosis. We have validated translocator protein as biomarker of breast cancer and established that dietary soy protein has a beneficial effect in delaying the development of breast cancer and controlling its aggressiveness. As PI on several NIH, DOD and FAMRI -funded research projects, I have trained a large number of postdoctoral (20), graduate (10), medical (24), dental (12), undergraduate (15) and high school students (8) in my laboratory. I have the expertise, leadership and enthusiasm necessary to participate in the summer research training of medical students as proposed in this project. I am member of Biochemical Society (London), American Institute of Biochemistry and Molecular Biology, Society of Sigma Xi, American Oil Chemists Society, Association of American Advancement of Sciences.
One or more keywords matched the following items that are connected to Das, Salil
Item TypeName
Academic Article Induction of neuronal damage in guinea pig brain by intratracheal infusion of 2-chloroethyl ethyl sulfide, a mustard gas analog.
Academic Article Antioxidant liposomes protect against CEES-induced lung injury by decreasing SAF-1/MAZ-mediated inflammation in the guinea pig lung.
Academic Article Effects of intratracheal exposure of 2-chloroethyl ethyl sulfide (CEES) on the activation of CCAAT-enhancer-binding protein (C/EBP) and its protection by antioxidant liposome.
Academic Article Signal transduction events in lung injury induced by 2-chloroethyl ethyl sulfide, a mustard analog.
Academic Article Prophylactic protection by N-acetylcysteine against the pulmonary injury induced by 2-chloroethyl ethyl sulfide, a mustard analogue.
Academic Article Evidence of hair loss after subacute exposure to 2-chloroethyl ethyl sulfide, a mustard analog, and beneficial effects of N-acetyl cysteine.
Academic Article Attenuation of half sulfur mustard gas-induced acute lung injury in rats.
Academic Article Decrease in brain POMC mRNA expression and onset of obesity in guinea pigs exposed to 2-chloroethyl ethyl sulfide, a mustard analogue.
Academic Article Inhibition of cholinephosphotransferase activity in lung injury induced by 2-chloroethyl ethyl sulfide, a mustard analog.
Academic Article Modulation of the expression of superoxide dismutase gene in lung injury by 2-chloroethyl ethyl sulfide, a mustard analog.
Academic Article Desensitization of beta-adrenergic receptors in lung injury induced by 2-chloroethyl ethyl sulfide, a mustard analog.
Academic Article Protection of half sulfur mustard gas-induced lung injury in guinea pigs by antioxidant liposomes.
Academic Article Role of MAPK/AP-1 signaling pathway in the protection of CEES-induced lung injury by antioxidant liposome.
Academic Article Activation of MAPK/AP-1 signaling pathway in lung injury induced by 2-chloroethyl ethyl sulfide, a mustard gas analog.
Concept Mustard Gas
Search Criteria
  • Mustard Gas
RCMI CC is supported by the National Institute on Minority Health and Health Disparities, National Institutes of Health (NIH), through Grant Number U24MD015970. The contents of this site are solely the responsibility of the authors and do not necessarily represent the official views of the NIH

For technical support please contact support